- Vasodilation and decreased fluid retention.
- Vasoconstriction, sodium and water retention, and cardiac remodeling, leading to worsening heart failure.
- Improved cardiac contractility.
- Decreased afterload.
161. A patient with heart failure experiences activation of the Renin-Angiotensin-Aldosterone System (RAAS). What is the technical pathophysiological consequence of RAAS activation in heart failure?
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