- Acute hyperglycemia.
- Damage to small blood vessels by chronic hyperglycemia.
- Hypoglycemia.
- Protein deficiency.
Author: ETEA MCQS.COM
No category found.
- Localized inflammation.
- Systemic manifestation of inflammation.
- Cellular adaptation.
- Genetic defect.
- Neutrophils.
- Macrophages and lymphocytes.
- Eosinophils.
- Basophils.
- Fibroblasts only.
- Myofibroblasts.
- Macrophages.
- Epithelial cells.
- Pigment accumulation.
- Protein accumulation.
- Lipid accumulation.
- Glycogen accumulation.
- Decreased oxygen demand.
- Pulmonary congestion and impaired gas exchange.
- Bronchospasm.
- Metabolic acidosis.
- Increased oxygen delivery to tissues.
- Anaerobic metabolism due to tissue hypoxia.
- Excessive bicarbonate production.
- Hyperventilation.
- Immediate IgE-mediated response.
- Antibody-mediated cell destruction.
- Immune complex deposition.
- Delayed T-cell mediated response.
- Blood glucose.
- White blood cell count.
- Hemoglobin level.
- Platelet count.
- Abnormal protein deposits in various organs.
- Excessive fat accumulation in the liver.
- Calcium deposits in soft tissues.
- Iron overload in the blood.
- Chromosomal abnormality.
- Single gene disorder.
- Multifactorial disorder.
- Environmental injury.
- Hypoparathyroidism.
- Malignancy and hyperparathyroidism.
- Chronic kidney disease.
- Vitamin D deficiency.
- Allodynia.
- Hyperalgesia.
- Central sensitization.
- Neuropathic pain.
- Coagulative necrosis.
- Liquefactive necrosis.
- Caseous necrosis.
- Fat necrosis.
- Widespread vasodilation and hypotension.
- Uncontrolled sympathetic overactivity below the level of injury in response to noxious stimuli.
- Bradycardia and warm, dry skin.
- Hypoglycemia.
- Fluid moving from the interstitial space into the intravascular space.
- Fluid moving from the intravascular space into the interstitial space (third spacing).
- Intracellular fluid moving into the extracellular space.
- Fluid moving from the extracellular space into the intracellular space.
