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No.	Topics.	No.	Topics.
1.	Adult health nursing -II	2.	Adult Health Nursing 1
3.	Anatomy & Physialogy 1	4.	Anatomy & Physiology -II
5.	Antomy	6.	Applied Nutrition
7.	Behavioral Psychology	8.	Bio chemistry for nurses
9.	Clinical Practicum	10.	Community Health Nursing -I
11.	Community Health nursing -III	12.	Community Health Nursing-I
13.	Community Health-II	14.	computer skills
15.	Critical Care Nursing	16.	Cultural Health & Society
17.	Developmental Pcychology	18.	English -VI ( Academic Writing )
19.	English 1 Fundamental English	20.	English VII Professional Writing
21.	English-II Communication Skills	22.	english-iii-technical-presentation-skills
23.	English-IV	24.	English-V
25.	Epidemoilogy	26.	Fundamenatals Of Nursing -II
27.	Fundamental of nursing	28.	Health Assessement 1
29.	Health Assessment -II	30.	Introduction To Biostatistics
31.	Introduction to Nursing Theories	32.	Islamiat
33.	Leadership and management in Nursing	34.	Mathematics
35.	Microbiology	36.	Mrental Health Nursinig
37.	Nursing Ethics	38.	Nursing Research
39.	Pak Study	40.	Pathophysiology
41.	Pathophysiology-II	42.	Pediartic Health Nursing-II
43.	Pharmacology -II	44.	Pharmacology-I
45.	Teaching Learning Principles & Practice

A patient with acute kidney injury (AKI) develops oliguria. This refers to:

Excessive urine output.
No urine output.
Decreased urine output.
Blood in the urine.

A patient with a genetic disorder has a mutated gene that leads to a deficiency in a lysosomal enzyme, causing accumulation of waste products in cells. This is an example of a:

Mitochondrial disorder.
Storage disorder.
Chromosomal abnormality.
Multifactorial disorder.

A patient with a severe bacterial infection develops uncontrolled activation of the coagulation cascade and fibrinolysis. This suggests:

Hemophilia.
Von Willebrand disease.
Disseminated Intravascular Coagulation (DIC).
Thrombocytopenia.

A patient with long-standing diabetes mellitus develops peripheral neuropathy. This is a microvascular complication affecting:

Large blood vessels.
Nerves.
Kidneys.
Eyes.

A patient with severe trauma develops hemorrhagic shock. The primary compensatory mechanism involves:

Decreased heart rate.
Vasodilation.
Increased systemic vascular resistance and heart rate.
Increased urine output.

A patient with a large abdominal wound that heals by “secondary intention” will likely experience:

Minimal scarring.
Significant granulation tissue formation and scarring.
Rapid closure with sutures.
No tissue loss.

A patient with chronic obstructive pulmonary disease (COPD) has a reduced FEV1/FVC ratio. This indicates:

Restrictive lung disease.
Obstructive lung disease.
Normal lung function.
Increased lung volume.

A patient with chronic heart failure has compensatory mechanisms that include activation of the Renin-Angiotensin-Aldosterone System (RAAS). This leads to:

Vasodilation and decreased fluid retention.
Vasoconstriction and fluid retention.
Decreased heart rate.
Increased myocardial contractility.

A patient with a Type IV hypersensitivity reaction (e.g., contact dermatitis) exhibits a delayed response. This delay is due to the time required for:

Antibody production.
Immune complex formation.
T-cell sensitization and migration.
Mast cell degranulation.

A patient with uncontrolled hypertension develops target organ damage. This is primarily due to:

Hypovolemia.
Persistent high pressure damaging blood vessels and organs.
Low blood glucose levels.
Enhanced renal function.

A patient with a severe infection develops the hallmark signs of inflammation: redness, heat, swelling, pain, and loss of function. The “redness” (rubor) is primarily caused by:

Vasoconstriction.
Increased blood flow (hyperemia).
Decreased blood flow.
Nerve damage.

A patient with a chronic inflammatory condition (e.g., rheumatoid arthritis) experiences morning stiffness that lasts for hours. This is a characteristic of:

Acute inflammation.
Chronic inflammation.
Resolution of inflammation.
Regeneration.

A patient with a head injury develops central diabetes insipidus. This is due to a deficiency in which hormone?

ADH (Antidiuretic Hormone).
Aldosterone.
Thyroid-stimulating hormone (TSH).
Cortisol.

A patient with Graves’ disease (hyperthyroidism) experiences symptoms like weight loss, tachycardia, and heat intolerance. This is caused by:

Decreased thyroid hormone production.
Autoimmune stimulation of thyroid hormone production.
Increased TSH levels.
Hypothalamic dysfunction.

A patient with iron deficiency anemia has fatigue and pallor due to:

Decreased oxygen-carrying capacity of the blood.
Increased white blood cell count.
Elevated platelet count.
Fluid retention.

A patient develops a pressure injury that appears as a deep crater with undermining and tunneling. This description aligns with which stage?

Stage I.
Stage II.
Stage III.
Unstageable.

A patient with a genetic predisposition and environmental triggers develops Type 2 Diabetes Mellitus. This is an example of which type of inheritance pattern?

Single gene disorder.
Chromosomal abnormality.
Multifactorial inheritance.
Mitochondrial inheritance.

A patient with acute myocardial infarction develops cardiac enzymes (troponin, CK-MB) in the bloodstream. These enzymes are released due to:

Normal cardiac function.
Myocardial cell necrosis.
Increased heart rate.
Decreased blood pressure.

A patient with a history of alcohol abuse presents with severe liver damage and bleeding esophageal varices. The bleeding is primarily due to:

Portal hypertension.
Decreased gastric acid.
Increased platelet count.
Enhanced clotting factors.

Category: BS Nursing

A patient with acute kidney injury (AKI) develops oliguria. This refers to:

A patient with a genetic disorder has a mutated gene that leads to a deficiency in a lysosomal enzyme, causing accumulation of waste products in cells. This is an example of a:

A patient with a severe bacterial infection develops uncontrolled activation of the coagulation cascade and fibrinolysis. This suggests:

A patient with long-standing diabetes mellitus develops peripheral neuropathy. This is a microvascular complication affecting:

A patient with severe trauma develops hemorrhagic shock. The primary compensatory mechanism involves:

A patient with a large abdominal wound that heals by “secondary intention” will likely experience:

A patient with chronic obstructive pulmonary disease (COPD) has a reduced FEV1/FVC ratio. This indicates:

A patient with chronic heart failure has compensatory mechanisms that include activation of the Renin-Angiotensin-Aldosterone System (RAAS). This leads to:

A patient with a Type IV hypersensitivity reaction (e.g., contact dermatitis) exhibits a delayed response. This delay is due to the time required for:

A patient with uncontrolled hypertension develops target organ damage. This is primarily due to:

A patient with a severe infection develops the hallmark signs of inflammation: redness, heat, swelling, pain, and loss of function. The “redness” (rubor) is primarily caused by:

A patient with a chronic inflammatory condition (e.g., rheumatoid arthritis) experiences morning stiffness that lasts for hours. This is a characteristic of:

A patient with a head injury develops central diabetes insipidus. This is due to a deficiency in which hormone?

A patient with Graves’ disease (hyperthyroidism) experiences symptoms like weight loss, tachycardia, and heat intolerance. This is caused by:

A patient with iron deficiency anemia has fatigue and pallor due to:

A patient develops a pressure injury that appears as a deep crater with undermining and tunneling. This description aligns with which stage?

A patient with a genetic predisposition and environmental triggers develops Type 2 Diabetes Mellitus. This is an example of which type of inheritance pattern?

A patient with acute myocardial infarction develops cardiac enzymes (troponin, CK-MB) in the bloodstream. These enzymes are released due to:

A patient with a history of alcohol abuse presents with severe liver damage and bleeding esophageal varices. The bleeding is primarily due to:

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