MCQs
6530 questions found
A.
Arterial spasm in the lungs.
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B.
A clot dislodges from a peripheral vein and travels to the pulmonary circulation.
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C.
Inhaled foreign body.
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D.
Direct lung infection.
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A.
Increasing gastric mucus production.
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B.
Inhibiting prostaglandin synthesis, which reduces gastric mucosal protection.
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C.
Stimulating gastric acid secretion.
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D.
Enhancing gastric blood flow.
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A.
Pallor, numbness, decreased temperature.
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B.
Redness, heat, swelling, pain, loss of function.
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C.
Jaundice, itching, weight loss.
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D.
Cyanosis, dyspnea, confusion.
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A.
IgE-mediated mast cell degranulation.
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B.
Antibody-dependent cellular cytotoxicity.
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C.
Formation of immune complexes that deposit in tissues, causing inflammation.
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D.
Delayed T-cell mediated response.
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A.
Impaired hemoglobin synthesis.
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B.
Abnormal chloride transport across cell membranes.
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C.
Autoimmune destruction of pancreatic cells.
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D.
Defective muscle protein.
✓
A.
Inflammation of the bladder only.
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B.
Chronic inflammation of the kidney parenchyma and renal pelvis.
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C.
Glomerular inflammation.
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D.
Formation of kidney stones.
✓
A.
Vasodilation to increase blood flow.
✓
B.
Activation of the sympathetic nervous system and renin-angiotensin-aldosterone system.
✓
C.
Decreased heart rate.
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D.
Increased urine output.
✓
A.
Arterial vasodilation.
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B.
Hardening and narrowing of arteries due to plaque buildup.
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C.
Venous inflammation.
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D.
Increased arterial elasticity.
✓
A.
Autoantibodies attacking the neuromuscular junction.
✓
B.
Systemic inflammation mediated by immune complexes and T-cells, primarily affecting synovial joints.
✓
C.
Destruction of pancreatic beta cells.
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D.
Increased red blood cell destruction.
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A.
Enhanced immune function.
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B.
Decreased blood glucose levels.
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C.
Impaired immune function and increased risk of infection.
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D.
Improved sleep patterns.
✓
A.
Normal scar formation.
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B.
Excessive collagen formation, extending beyond the original wound boundaries.
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C.
Incomplete wound closure.
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D.
Decreased tensile strength.
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B.
Hemoglobin synthesis.
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C.
Platelet production.
✓
B.
Massive pulmonary edema and severe hypoxemia due to capillary-alveolar membrane damage.
✓
C.
Chronic airway inflammation.
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D.
Alveolar wall destruction.
✓
A.
Increased capillary hydrostatic pressure.
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B.
Decreased plasma oncotic pressure.
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C.
Increased lymphatic drainage.
✓
D.
Increased arterial blood flow.
✓
B.
Secondary intention.
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A.
Alveolar wall destruction.
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B.
Airway inflammation, mucus gland hyperplasia, and excessive mucus production.
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C.
Reversible bronchospasm.
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D.
Decreased peripheral airway resistance.
✓
A.
The heart pumps blood efficiently.
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B.
The heart's pumping ability is decreased.
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C.
The heart valves are narrowed.
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D.
The patient is experiencing fluid volume deficit.
✓
