MCQs

β-oxidation releases acetyl-CoA and reducing equivalents.

Fat reserves become the major source of energy.

Both glucose and fatty acids ultimately produce acetyl-CoA.

Deamination removes the amino group before respiration.

β-oxidation repeatedly cleaves fatty acids into acetyl-CoA units.

Glycerol enters glycolysis, fatty acids yield acetyl-CoA for Krebs cycle.

Acetyl-CoA is the central substrate for the Krebs cycle.

Amino groups must first be removed.

Greater hydrogen content provides more electrons for ATP generation.

Protein catabolism ultimately feeds into respiratory pathways.

Carbon skeletons enter various points of cellular respiration.

β-oxidation repeatedly removes two-carbon acetyl-CoA units.

Glycerol enters glycolysis after conversion into suitable intermediates.

Lipases hydrolyze triglycerides into glycerol and fatty acids.

Fatty acids undergo β-oxidation producing acetyl-CoA, which enters the Krebs cycle.

Loss of helper T cells severely compromises immune function.