β-oxidation releases acetyl-CoA and reducing equivalents.
Fat reserves become the major source of energy.
Both glucose and fatty acids ultimately produce acetyl-CoA.
Deamination removes the amino group before respiration.
β-oxidation repeatedly cleaves fatty acids into acetyl-CoA units.
Glycerol enters glycolysis, fatty acids yield acetyl-CoA for Krebs cycle.
Acetyl-CoA is the central substrate for the Krebs cycle.
Amino groups must first be removed.
Greater hydrogen content provides more electrons for ATP generation.
Protein catabolism ultimately feeds into respiratory pathways.
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