β-oxidation releases acetyl-CoA and reducing equivalents.
Protein catabolism ultimately feeds into respiratory pathways.
Greater hydrogen content provides more electrons for ATP generation.
Amino groups must first be removed.
Acetyl-CoA is the central substrate for the Krebs cycle.
Glycerol enters glycolysis, fatty acids yield acetyl-CoA for Krebs cycle.
β-oxidation repeatedly cleaves fatty acids into acetyl-CoA units.
Fatty acids undergo β-oxidation producing acetyl-CoA, which enters the Krebs cycle.
Lipases hydrolyze triglycerides into glycerol and fatty acids.
Glycerol enters glycolysis after conversion into suitable intermediates.
β-oxidation repeatedly removes two-carbon acetyl-CoA units.
Carbon skeletons enter various points of cellular respiration.
Chronic infection often causes fatigue.
HIV is an acellular virus.
ART reduces viral multiplication.
Early treatment greatly improves survival and quality of life.
Loss of helper T cells severely compromises immune function.
Viral load measures HIV quantity in blood.
Reduced immunity permits fungal growth.
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