MCQs
6530 questions found
A.
Autoimmune inflammation.
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B.
Deposition of uric acid crystals in the joint, leading to an inflammatory response.
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C.
Bacterial infection.
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D.
Degeneration of articular cartilage.
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A.
Oral antibiotics and observation.
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B.
Immediate administration of broad-spectrum antibiotics, intravenous fluids, and vasopressors to restore tissue perfusion.
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D.
Oxygen therapy only.
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A.
Reduced renal perfusion.
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B.
Direct damage to kidney tubules.
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C.
Obstruction of urinary outflow from the kidneys.
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D.
Glomerular inflammation.
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B.
Increased bicarbonate levels (e.g., due to vomiting or diuretic use) or excessive acid loss.
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C.
Decreased production of lactic acid.
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D.
Excessive loss of CO2?.
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B.
Decreased intravascular volume, leading to reduced preload, cardiac output, and tissue perfusion.
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C.
Increased cardiac contractility.
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D.
Increased systemic vascular resistance.
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A.
Localized inflammation.
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B.
Systemic inflammatory response to pancreatic enzymes, leading to widespread organ damage.
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A.
Increased tubular reabsorption.
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B.
Glomerular basement membrane thickening and increased permeability.
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C.
Renal artery stenosis.
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D.
Urinary tract infection.
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A.
Decreases blood pressure.
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B.
Breaks down fibrin clots, restoring blood flow to ischemic brain tissue.
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C.
Prevents platelet aggregation.
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D.
Increases cerebral blood flow by vasodilation.
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A.
Decreased production of lactic acid.
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B.
Excessive elimination of CO2? from the lungs.
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C.
Increased bicarbonate reabsorption.
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D.
Overproduction of ketone bodies.
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A.
Loss of motor function.
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B.
Exaggerated sympathetic response to noxious stimuli below the level of injury, due to impaired autonomic regulation.
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C.
Peripheral nerve damage.
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D.
Brainstem compression.
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A.
To treat symptomatic UTI.
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B.
To identify and urgently treat pyelonephritis (kidney infection) to prevent sepsis and kidney damage.
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C.
To advise increased fluid intake.
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D.
To perform a urinalysis.
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B.
Inflammation of the glomeruli, leading to increased permeability and impaired filtration.
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C.
Obstruction of the urinary tract.
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D.
Renal artery stenosis.
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B.
Accumulation of uremic toxins.
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A.
To administer more warfarin.
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B.
Immediate reversal of anticoagulation with Vitamin K and/or fresh frozen plasma (FFP).
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C.
To administer aspirin.
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D.
To observe the bleeding.
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A.
To increase blood viscosity.
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B.
To maintain tissue oxygen delivery despite reduced oxygen-carrying capacity.
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C.
To decrease cardiac workload.
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D.
To increase red blood cell production.
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A.
Inflammation of the pancreas.
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B.
Erosion of the gastric or duodenal mucosa by acid and pepsin.
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A.
Diastolic dysfunction.
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B.
Impaired systolic function and reduced cardiac output.
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D.
Normal cardiac function.
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A.
Decreased calcium absorption.
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B.
Excess cortisol leading to increased bone resorption and decreased bone formation.
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C.
Vitamin D deficiency.
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D.
Increased estrogen levels.
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A.
To lower blood pressure slowly over days.
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B.
To reduce blood pressure promptly but carefully to prevent end-organ damage.
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C.
To administer oral diuretics only.
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D.
To observe without intervention.
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